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Essay / Research Paper Abstract
A 4 page discussion of the benefits and potential side effects and contraindications of these two drugs. The author provides a side by side comparison of the pharmacological mechanisms of these drugs and concludes which is most suitable in specific situations. Bibliography lists 4 sources.
Page Count:
4 pages (~225 words per page)
File: AM2_PPtomoxi.rtf
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Unformatted sample text from the term paper:
Arimidex and Tamoxifen have been the pharmacological backbone of breast cancer treatment since their introduction. The anti-estrogen drug Tamoxifen, in particular, has
been widely employed for over thirty years (Obesity, Fitness & Wellness Week, 2005). Arimidex, in contrast, is a relative newcomer on the scene. It is the only aromatase
inhibitor, however, that has been used very extensively in fighting breast cancer. Both Tamoxifen and Arimidex, unfortunately, are associated not only with clinical success but also with certain side
effects and contraindications. Arimidex seems superior to Tamoxifen, however, in terms of the risks that it presents in regard to the development of endometrial cancer among postmenopausal women (Womens
Health Weekly, 2002). Arimidex is now believed, in fact, to be more effective in the prevention of the recurrence of breast cancer as well in post-menopausal women (Obesity, Fitness
& Wellness Week, 2005). It is not utilized in younger women, however (Obesity, Fitness & Wellness Week, 2005). Tamoxifen is
an anti-estrogen. In other words its pharmaceutical action centers around the prevention of estrogen bonding to the bodys cells (Obesity, Fitness & Wellness Week, 2005). This is important
because estrogen is associated with the development of an estimated three-fourths of post-menopausal tumors (Obesity, Fitness & Wellness Week, 2005). Long term use has witnessed a fifty percent
reduction in breast cancer recurrence for those women that use it (Obesity, Fitness & Wellness Week, 2005). Arimidex, in comparison, is a aromatase inhibitor (Obesity, Fitness & Wellness Week,
2005). Rather than acting to prevent recurrences of breast cancer by blocking the adhesion of estrogen to the cell, it prevents the production of estrogen in the first place
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