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article is about the extent to which stem cell biology is ever-improving its component as a system of self-renewal, with particular importance to the study and identification of cancer cells.
The authors point out how significant similarities exist between stem and cancer cells inasmuch as mutated (cancerous) cells are derived from healthy cells, thereby giving scientists reason to surmise
that cancer cells may have the capacity for regeneration through rare cancer stem cells. B. Kogan, S. C.; et al (2001). BCL-2 cooperates with promyelocytic leukemia retinoic acid receptor
chimeric protein (PMLRAR ) to block neutrophil differentiation and initiate acute leukemia. J. Exp. Med. 193, 531-543. Reya et als article cited
this article to support the claim that suppressed apoptosis (cell self-destruction) can beneficially impact tumorigenesis (tumor production) through cellular changes. C. Domen, J. & Weissman, I. L. (2000). Hematopoietic
stem cells need two signals to prevent apoptosis; BCL-2 can provide one of these, Kitl/c-Kit signaling the other. J. Exp. Med. 192, 1707-1718.
Reya et als article cited this article to support the claim that apoptosis (cell self-destruction) caused by growth factor deprivation can be protected by two individual signals that result
in differentiation rather than self-renewal. D. Weissman, I. L. (2000). Translating stem and progenitor cell biology to the clinic: barriers and opportunities. Science 287, 1442-1446.
Reya et als article cited this article to support the claim that the extensive and growing regenerative capacity of stem cells in relation to other systems
of the body has been instrumental in realizing the potential for cancer stem cells to be used as a defense against the cancer cell.
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