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Essay / Research Paper Abstract
A 6 page overview of the manner in which individuals from high altitude cultures function in high altitude situations. The author discusses the importance of hematological changes, differences in chest morphology, pulmonary function, and metabolism that characterize these people. Bibliography lists 16 sources.
Page Count:
6 pages (~225 words per page)
File: AM2_PPaltitu.rtf
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Unformatted sample text from the term paper:
physiologists that those that lived in high altitudes were physiologically compromised, that the so-called "rarefied" air that they breathed combined with the low barometric pressure and high elevation to result
in lowered oxygen availability and consequently "physical lethargy and cerebral anemia" (de Romo and Padilla, 2003). We now know that despite the fact that modern science has validated some
of these beliefs, those that live in high altitudes frequently enjoy physiological adaptations that allow them to function in those altitudes without sever physiological impact.
High altitudes such as those found on the Andean, Tibetan, and east African plateaux, does indeed introduce certain characteristic stressors to these regions inhabitants (Beall, 2003). Beall
(2003) observes, for example, that one breath at sea level contains 62 percent oxygen while one breath at an altitude of 4000 m contains only 20.93 percent oxygen. Those
accustomed to living at low altitudes are at a definite disadvantage under these lower oxygen concentrations. Those individuals that are accustomed to lower elevations can begin to experience
the impacts of hypoxia at around 2,500 meters elevation (Jansen, Krins, and Basnyat, 1999; Barry and Pollard, 2003; Aldenderfer, 2003). Acute
mountain sickness (AMS)is one of the more common illnesses that inflict travelers to high altitudes (Jansen, Krins, and Basnyat, 1999). At extreme altitudes healthy individuals find themselves in a
situation which normally would be typified as respiratory failure. Without acclimatization AMS may develop and this can lead to high-altitude pulmonary edema (HAPE) as well as high-altitude cerebral edema
(HACE) (Jansen, Krins, and Basnyat, 1999). Both HAPE and HACE are potentially lethal (Jansen, Krins, and Basnyat, 1999). Jansen, Krins, and Basnyat (1999) explain that, for lowlanders that
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