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Essay / Research Paper Abstract
A 4 page overview of this bone disorder. Associated with Vitamin D deficiencies, the condition is most often successfully treated
with simple Vitamin D supplementation. Bibliography lists 5 sources.
Page Count:
4 pages (~225 words per page)
File: AM2_PPosteom.rtf
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Unformatted sample text from the term paper:
cannot absorb sufficient enough dietary calcium, a mineral that is critical to bone strength and density (Drug Week, 2004). Osteomalacia can result from environmental or genetic causes. Interestingly,
however, Drug Week (2004b, 411) reports: "Vitamin D receptor gene polymorphisms dont associate with osteomalacia"
Oncogenic osteomalacia, in contrast, is a tumor associated form of the condition (Science Letter, 2005).
Osteomalacia is characterized by the weakening and softening of bones and even their deformation or fracture (Drug Week, 2004). It is more common among the elderly and those
of Asiatic descent (Womens Health Weekly, 2005). The healthy skeleton is in process of continually breaking down but this breakdown
is offset by a corresponding buildup. Specialized bone cells called osteoclasts are responsible for the destruction of bone cells. This destroyed osseous tissue is reabsorbed by the body
and other specialized cells called osteoblasts create new bone to fill in where the old cells were taken out. In a healthy individual this process typically takes about three
months from start to completion. This process takes place through mitotic division. The exception, however, is that the normal bone cells filling in the gaps are not malignant,
they are healthy cells whose job it is to constantly replenish the skeletal system. This process is not limited to the latter years of an individuals life, however.
It is one which occurs throughout life and in addition to simple cell replacement it is also responsible for the repair of damaged cells which are incurred during fractures.
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