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Essay / Research Paper Abstract
A 4 page research paper that discusses nutrition and muscular degenerative disorders. The writer argues that the neurological disorders that the student is investigating, i.e., Guillian-Barre (GB), myasthenia gravis (MG), Parkinson's disease (PD), spinal cord injury (SCI) and multiple sclerosis (MS), have the common factor, from a nutritional point of view, of entailing the danger of developing myopathies. In each of these cases, and for a variety of physiological reasons, muscle protein depletion occurs at a faster pace and is far more extensive than would normally occur from inactivity (Burnham, Moss and Ziegler, 2005). Nutritional implications are discussed. Bibliography lists 5 sources.
Page Count:
4 pages (~225 words per page)
File: D0_khnutdis.rtf
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Unformatted sample text from the term paper:
the danger of developing myopathies. In each of these cases, and for a variety of physiological reasons, muscle protein depletion occurs at a faster pace and is far more extensive
than would normally occur from inactivity (Burnham, Moss and Ziegler, 2005). Novel nutritional therapies have been developed for these patients, "such as glutamine and glutathione supplementation," which are designed
to counteract, at least to a certain extent, associated myopathy (Burnham, Moss and Ziegler, 2005, p. 1818S). Glutamine (GLN) is a part of more metabolic processes than any other amino
acid, with GLN produced and warehoused in skeletal muscle and it is responsible for both stimulating protect synthesis and inhibiting protein breakdown (Burnham, Moss and Ziegler, 2005). Nevertheless, despite
the crucial role that glutamine plays in critical illness, in general, it is not present in commercially available "conventional patenteral nutrition solutions," and, furthermore, it is present in only modest
amounts in the majority of commercially prepared "enteral nutrient products" (Burnham, Moss and Ziegler, 2005, p. 1818S). While research has definitely identified a crucial role for glutamine, studies also suggest
that the loss of amino acids and muscle mass fail to adequately account for the full extent of critical illness myopathy and there is evidence that "disturbances in electrolytes and
the calcium gradients of the muscle" also plays a role in loss of muscle contractility (Burnham, Moss and Ziegler, 2005, p. 1818S). The immobility that results from a critically
illness, such as GB, contributes to "decreased gastric motility, dysphagia, and depression, which all contribute to malnutrition (Sulton, 2002, p. 25). The danger of aspiration further complicates the patients ability
to eat and it is imperative, therefore, that the pharyngeal function should be routinely monitored (Sulton, 2002). Patients who cannot swallow will require either enteric or parenteral nutrition (Sulton, 2002).
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