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Essay / Research Paper Abstract
This 6 page paper provides an overview of the impacts of measles infection and the nature of the immune response. Bibliography lists 4 sources.
Page Count:
6 pages (~225 words per page)
File: MH11_MHMeasl.rtf
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Unformatted sample text from the term paper:
the respiratory mucosa, there are both innate and adaptive immune protection processes that this virus will encounter at the mucosal surface. Initially, innate systems are at work, including the presence
of sticky mucus in the respiratory track can trap the virus in secretions that will be expectorated. The virus could also be encounter macrophages, which will serve as a
warning system for antigen presenting cells (APCs) in the underlying immune tissues (Goldsby, 2002). Adaptive immune protection, then, comes into play after macrophages have consumed some of the attacking
virus and subsequent T-cells become sensitized to the proteins displayed by the macrophages. Though less of this takes place at the mucosal surface (and more at the underlying immunity
level), it is important to note the potential presence of macrophages at the mucosal surface and the presence of cell-mediated immunity that corresponds with the use of vaccinations or previous
exposure to the virus (Goldsby, 2002). 2) As this is a virus, would you expect an antibody response, a cellular response, or both. Why? The basis of
determining whether a response to the presence of the measles virus would be an antibody response or a cellular response is dependent on whether this is a persons first exposure
to the virus or whether they have had a vaccination or previous exposure to the virus. If a vaccination or a previous exposure is present, an antibody response can be
expected because of the existence of sensitized T-cells. If the person has not been exposed to measles in the past, then a cellular response would be expected, with the
most significant cellular response being that of macrophages, which would consume the invading virus cells and display their proteins as the basis for the sensitization of the T-cells.
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