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Essay / Research Paper Abstract
This 16 page paper addresses major health issues. The problem of hypertension is widespread in the United States, with more than half of the people in the nation developing confirmed cases in the course of their lifetimes. In fact, the disease itself and the correlated problems, including the development of multiple atherosclerotic diseases, the leading causes of morbidity and mortality in the United States, has been defined as major health concerns and the focus of significant study. Bibliography lists 10 sources.
Page Count:
16 pages (~225 words per page)
File: MH11_MHHYPVMC.RTF
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the disease itself and the correlated problems, including the development of multiple atherosclerotic diseases, the leading causes of morbidity and mortality in the United States, has been defined as major
health concerns and the focus of significant study (Kaplan, 1996). "Most of the predominately systolic hypertension that appears after 60 years of age can be directly attributed to atherosclerotic encroachment
of the larger capacitance arteries, causing systolic pressures to rise inordinately when cardiac output enters a rigid, inelastic vascular bed. In the 15% of people with both high systolic and
diastolic pressures that appear earlier in life--the disease called essential hypertension --atherosclerosis is accelerated and is responsible for most of the eventual complications" (Kaplan, 1996, p. 1919).
Molecular Pathophysiology Preventative treatment options for patients suffering from hypertension and correlated cardiac conditions are based in a knowledge of the normal cellular function, cell abnormalities and reactive processes
that are inherent within the hearts of individuals who have suffered from heart failure. Clinical syndromes of heart failure have been considered in order to outline the abnormalities in
gene expression associated with the development of atherosclerosis, as well as the chronic stages that initiate molecular failure, and the abnormalities in the cellular makeup that result in the development
of atherosclerosis, and the progression of correlated hypertension and myocardial dysfunction (Katz, 1990). The pursuit of continued exploration of cellular processes involved in hypertension and atherosclerosis is imperative to
successes in reducing the deterioration of the circulatory process that can inherently result in necrosis of the cells in the heart muscle (Katz, 1990). As a result, the pursuit
of an understanding of the molecular biology and Pathophysiology of the arteries and heart and the progression of myocene failure, gene expression and myocardial repair are necessary elements in determining
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