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Essay / Research Paper Abstract
This is a 5 page paper that provides an overview of mad cow disease. Variants of the disease as well as potential treatments are explored. Bibliography lists 6 sources.
Page Count:
5 pages (~225 words per page)
File: KW60_KFbio001.doc
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Unformatted sample text from the term paper:
no means consistent. All too frequently, new diseases have the potential to arise which confound the understanding of medical science, and which represent a potentially major concern for epidemiologists who
may be unsure about the best or most appropriate methods for containing the disease. For instance, one might look to the avian flu or swine flu epidemics of recent years
for an example of a disease that caused concern due to its rapid spread through congested populations without any fully understood way of curing the disease or preventing the spread.
However, one of the most impactful of all diseases to appear in the 20th century is bovine spongiform encephalopathy, of "mad cow disease". A disease with the major potential to
epidemically spread through the food supply, and with no known cure, it represents a major obstacle for disease management going into the 21st century. This paper will explore how mad
cow disease affects human physiology, and recent advancements in treatment and containment technologies. This paragraph helps the student begin to explore the particulars of mad cow disease. TO begin with,
it is necessary to have a brief understanding of what mad cow disease is, and how it tends to spread to the human population. While the disease is called "mad
cow disease", it is obviously by no means confined to the bovine organism; rather, the fundamental nature of the disease is that it is caused by infected prions, or proteins
that can be viably passed between species (Helmenstein, 2012). The name comes from the fact that humans tend to get the disease from eating infected beef manufactured from cows who
are, in turn, infected from eating feed that has been "enhanced" with protein from infected animal sources, such as sheep (Helmenstein, 2012). The capacity of the disease to affect the
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