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Essay / Research Paper Abstract
This 5 page paper discusses the disease non-hodgkins lymphoma with regard to anti-apoptosis. Bibliography lists 5 sources.
Page Count:
5 pages (~225 words per page)
File: KV32_HVlenert.rtf
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Unformatted sample text from the term paper:
with cancer patients, the best place to start seemed to be with a definition. According to a medical dictionary, "diffuse mixed cell non-Hodgkins lymphoma" is given as the definition of
a condition called "Lennarts lymphoma," a "malignant lymphoma with high content of epithelioid cells" (Segen, 1992). Because that dictionary definition is literally the only response to the inquiry, it seems
logical to use "Lennarts lymphoma" as the search term from here on. Next, it is necessary to define "anti-apoptosis." Apoptosis is the natural process of cell death. Its important
that cells die off to make room for new ones and this process, which is also known as "programmed cell death" is the mechanism by which they do so. Since
cancer is unregulated cell growth, it would seem that apoptosis would be a natural adjunct to any other treatment modalities. Anti-apoptosis, then, is anything that interferes with the process of
cell death; this suggests that in dealing with cancer, inhibiting the cellular death process would be detrimental to the patient, who wants to stop the growth of cells. A recent
article deals with the identification of molecular subgroups classified as peripheral T-cell lymphomas (PTCL) (Ballester et al, 2006). To establish the molecular classification "PTCL," the authors analyzed 59 "primary nodal
T-cell lymphomas using cDNA microarrays, including 56 PTCL and three T-lymphoblastic lymphoma (T-LBL)" (Ballester et al, 2006, p. 1560). The expression profiles were able to discriminate between "angioimmunoblastic lymphoma, anaplastic
large-cell lymphoma and T-LBL" (Ballester et al, 2006, p. 1560). The researchers could separate PTCL-U into "three molecular subgroups called U1, U2 and U3" by the use of a multiclass
predictor; they give the definitions of all three subgroups, but the one of interest here is U2, which was associated with "overexpression of genes involved in T-cell activation and apoptosis"
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