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Essay / Research Paper Abstract
An overview of the phenomenon of ageing from a biochemical perspective, looking at the programmed and random damage theories and the ways in which these might be approached in order to take into account both genetic and stochastic causes of ageing. Bibliography lists 5 sources
Page Count:
5 pages (~225 words per page)
File: JL5_JLageing.rtf
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Unformatted sample text from the term paper:
a complex one, and one which must therefore be addressed from a number of different perspectives if a cohesive analysis of the phenomenon as a whole is to be achieved.
Ageing can be observed to manifest itself in numerous organic systems: CNS, endocrine, cardiovascular and immune systems, for example, are all affected.
The endocrine system, for instance, undergoes changes due to the changes in the way
that hormones are synthesised, in the functioning of hormone receptors, and the premature degeneration of hormone-producing cells: these changes can give rise to various age-related conditions such as thyroid dysfunction,
diabetes and changes in the levels of sex hormones.
The immune system becomes less efficient with age, the decreased functionality of T cells meaning that the system is less able to cope with infection. Effross
(2003) study found that T cells repeated stimulated with antigen reached a point of cell cycle arrest; they were no longer able to proliferate, and this is supported by the
increased number of T cells with identical phenotypes which are found in the elderly.
Effects on the cardiovascular system include increased heart size and decrease in the elasticity of blood vessels, which leads to an increase in
blood pressure concomitant with the ageing process.
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